If the client has decreasing blood pressure after the surgery, there is a chance that the kidney will sense a decreased in circulating blood volume. When this happen, immediately an angiotensin I is released by the kidney, this will follow with the production of angiotensin II. This is known as a powerful vasoconstrictor. Additionally, it will stimulate the adrenal cortex to release aldosterine which can cause active reabsorption of sodium and water.
The baroreceptors located in the aortic arch of the heart and carotid sinus will answer to altered arterial pressure which leads to the initiation of events that ultimately stimulate peripheral vasoconstriction, as a result, an output of increasing cardiac
