Answer:
The correct answer is option A. a mutation in the Z-disk protein that caused the attachment of the Z disk to the titin protein to be stronger
Explanation:
As delayed onset muscle soreness is thought to be led by the inflammatory response of the body in order to repair damage to the Z disks and contractile filaments in muscles, due to the intense exercise.
Mutation in the Z-disk protein would strengthen the connection between Z disks and contractile filaments as it is logical that this mutation could withstand more pressure or intense exercise before damage occurs to these tissues.