Question 2 On 11/2 we did a case study about cyanide poisoning and how it directly affects cellular respiration. Here, consider instead a poison that causes the immediate oxidation of NADH to NAD+ in the mitochondrial matrix. Please answer these three questions: a) How would this poison affect the usual workings of cellular respiration (including the electron transport chain)? b) Would oxygen consumption go up or down? c) How would ATP production change? (Would it be the same, go to zero, or be somewhere in between?) What is your reasoning? If you answer"in between", how many ATP molecules per glucose do you hypothesize would be made? What is your reasoning?